Written in English
|Statement||by Mara Tema Slawsky.|
|The Physical Object|
|Pagination||viii, 131 leaves|
|Number of Pages||131|
Journals & Books; Help When repeatedly elicited, the oscillatory escape swim of the marine molluscTritonia diomedeaundergoes habituation of the number of cycles per swim. Previous work has shown that this habituation is accompanied by sensitization of another feature of the behavior: latency to swim onset. Presynaptic inhibition in the Cited by: Here, in the marine mollusk Tritonia diomedea, we describe a detailed cellular mechanism for PPI--a combination of presynaptic inhibition of startle . The mollusk Tritonia diomedea is a good model to investigate the behavioral and neural responses to the magnetic field. The mollusk inhibits all . Here, in the marine mollusk Tritonia diomedea, we describe a detailed cellular mechanism for PPI—a combination of presynaptic inhibition of startle afferent neurons together with distributed postsynaptic inhibition of several downstream interneuronal sites in the startle circuit.
Slawsky M. Presynaptic inhibition in the marine mollusk Tritonia Diomedea. Palo Alto: Stanford University; Smith KJ, McDonald WI. The pathophysiology of multiple sclerosis: the mechanisms underlying the production of symptoms and the natural history of the disease. Philos Trans R Soc Lond B Biol Sci. ; – Tritonia diomedea is a marine nudibranch mollusk attractive for neurophysiological studies because of its large pigmented neurons, many of which are individually identifiable from animal to animal. Upon skin contact with its seastar predators, Tritonia launches a rhythmic escape swim consisting of a series of alternating ventral and dorsal. Tritonia, may be important for self-reconfiguration of the swim motor network. [Key words: 5-HT, heterosynaptic facilitation, nudibranch mollusk, presynaptic mechanism, neuromodulation, cen- tral pattern generator] Heterosynaptic regulation of neurotransmitter release allows a level of fine control for neuronal circuits. Here, we demonstrate that in the marine mollusk Tritonia diomedea, the serotonergic dorsal swim interneurons (DSIs) heterosynaptically increase the .
Here, in the marine mollusk Tritonia diomedea, we describe a detailed cellular mechanism for PPI--a combination of presynaptic inhibition of startle afferent neurons together with distributed postsynaptic inhibition of several downstream interneuronal sites in the startle circuit. PMID: [Indexed for MEDLINE] Publication Types. It has been know for three decades that the marine mollusk Tritonia diomedea has an ability for geomagnetic orientation (Lohmann and Willows ), . Popescu IR, Frost WN () Highly dissimilar behaviors mediated by a multifunctional network in the marine mollusk Tritonia diomedea. J Neurosci Sakurai A, Darghouth NR, Butera RJ, Katz PS () Serotonergic enhancement of a 4-AP-sensitive current mediates the synaptic depression phase of spike timing-dependent . INTRODUCTION. The marine mollusc Tritonia diomedea initiates a rhythmic escape swim upon contact with a sufficiently aversive stimulus, such as the seastar Pycnopodia helianthoides ().The neural program underlying this behavior can be elicited by briefly stimulating any one of a number of peripheral nerves in the isolated brain preparation, indicating that the motor program is .